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Immunology (Neutrophil) Lecture 3 Part 8

Immunology (Neutrophil) Lecture 3 Part 8

Immunology (Neutrophil) Lecture 3 Part 7

Immunology (Neutrophil) Lecture 3 Part 7

Immunology (Neutrophil) Lecture 3 Part 9

Immunology (Neutrophil) Lecture 3 Part 9

Immunology (Neutrophil) Lecture 3 Part 6

Immunology (Neutrophil) Lecture 3 Part 6

Modulation of pro-inflammatory and regulatory NK cell responses. DCs infected with intracellular bacteria produce IL-12. (A) In localized infections, NKs respond to IL-12, producing IFN-γ, which ++ the DC IL-12 response, creating an unregulated feedback loop. The pathogen is killed (with tissue damage). (B) In systemic infection, differences in the environmental accessory cells (concentration of IL-12?) lead to NK production of IL-10: limits the damage but may also allow the infection to…

Modulation of pro-inflammatory and regulatory NK cell responses. DCs infected with intracellular bacteria produce IL-12. (A) In localized infections, NKs respond to IL-12, producing IFN-γ, which ++ the DC IL-12 response, creating an unregulated feedback loop. The pathogen is killed (with tissue damage). (B) In systemic infection, differences in the environmental accessory cells (concentration of IL-12?) lead to NK production of IL-10: limits the damage but may also allow the infection to…

Immunology (Neutrophil) Lecture 3 Part 5

Immunology (Neutrophil) Lecture 3 Part 5

https://resources.rndsystems.com/images/site/MP_Inflammasomes_1550-header.png

https://resources.rndsystems.com/images/site/MP_Inflammasomes_1550-header.png

Every toll-like receptor (TLR) in mammals has the ability to identify pathogens based on their distinct molecular configuration.

Every toll-like receptor (TLR) in mammals has the ability to identify pathogens based on their distinct molecular configuration.

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